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If it were ventricular fibrillation you wouldn't be making any recordings. You'd be face down on the floor, dead. It's impossible to be conscious during v-fib. That entire first strip is just noise and should be ignored. In fact, almost all recordings from consumer grade devices like Kardia, Apple Watch, etc. show mainly a bunch of noise in the first few seconds of recording, so doby Carey - AFIBBERS FORUM
Lots of people make that choice. They're in afib constantly but they experience few if any symptoms, so they take an anticoagulant to prevent strokes and maybe a rate control drug to keep their HR in check, and then they're good. Their life expectancy is the same as anyone else's, so why do anything more? I know several such people, a couple of whom are serious runners and cyclistsby Carey - AFIBBERS FORUM
Ever had a sleep study done? What you're describing could be sleep apnea.by Carey - GENERAL HEALTH FORUM
Officially, my Watchman was implanted by Natale, but both Horton and Natale were present. At the time (2018), Natale had done something like 500 Watchman implants while Horton had done thousands. If I needed to have someone poking around in my LAA, Horton would be my first choice.by Carey - AFIBBERS FORUM
You're rate's not super high, so you can put up with it for quite a while, but I wouldn't let it go more than a week or two. But that should be plenty of time for you to arrange it with your EP. I doubt an ER would cardiovert a rate that low.by Carey - AFIBBERS FORUM
I'm not questioning your cardiologist's experience. I'm questioning her training for your specific problem. I'm sure she's a fine cardiologist but she simply doesn't have the training to deal with afib that an EP does. We see this all the time here. General cardiologists just generally don't follow current medical standards for the treatment of afib because theyby Carey - AFIBBERS FORUM
If you start amiodarone, you'll have to come right back off it very soon for an ablation at the end of the year, so I don't think that's a viable option. Were you on the sotalol before your last cardioversion? If not, then it's worth doing another one. All the drugs you mentioned are better at keeping you out of flutter than they are converting it. I dealt with intractableby Carey - AFIBBERS FORUM
As of this date, no posts may be made on this forum regarding the following subjects: COVID-19 COVID-19 treatments Vaccines Conspiracy theories Libelous comments aimed at any individual Claims of scientific fact without supporting evidence from credible sources If you have a question or comment about any of those subjects that's relevant to atrial arrhythmias or cardiology in genby Carey - GENERAL HEALTH FORUM
No, not really. I suppose general cardiovascular fitness could only help but I don't know of anything you can do to specifically target the LAA.by Carey - AFIBBERS FORUM
Ice Man, is your cardiologist also an electrophysiologist (EP)? EPs are cardiologists who specialize in heart rhythm problems, and that's who you need to be dealing with. It sounds like you're seeing a general cardiologist because it's really unlikely an EP would tell you to just take metoprolol if your afib returns. Metoprolol doesn't prevent afib, so it's not a solutionby Carey - AFIBBERS FORUM
This is the problem with sites like drugs.com. Although the information is correct, it's so abbreviated that you almost need an MD to interpret it correctly. The warnings drugs.com is talking about don't apply to you. They only apply to people with a history of heart disease, heart attack and/or heart failure in particular. In treating afib, flecainide is almost always combined with metby Carey - AFIBBERS FORUM
You're on the maximum dosages of both drugs. Talk to your EP and ask about cutting the dosages down because of the side effects. I would ask to cut both in half, and then adjust later if needed.by Carey - AFIBBERS FORUM
Yeah, but fatigue shows up as a side effect of just about every drug on the planet. I've taken diltiazem and it caused significant fatigue for me. Not as bad as beta blockers, but enough to reduce my cycling abilities. Flecainide did not.by Carey - AFIBBERS FORUM
Quotecolindo Just to turn things around a bit, You seem to be inferring that nattokinase is inferior to Eliquis, that may be so but where is the proof, who can say for sure? can you? No, I don't know that nattokinase is inferior. I also don't know that it's superior. That's because it's never been subjected to clinical trials comparing them, so we really don't knby Carey - AFIBBERS FORUM
I doubt it's the flecainide. It's more likely the diltiazem. Diltiazem is known to cause fatigue like that. You didn't mention the dosage, but lowering it would probably help. And not that it really matters, but your ablation didn't fail. An ablation that lasts one year is considered successful, and there's no way for that success to be undone. What happened is your afby Carey - AFIBBERS FORUM
Yes, that's pretty standard procedure for most EPs. They want your heart as susceptible to afib as possible during the procedure.by Carey - AFIBBERS FORUM
I don't see how the two things are connected. I would get that mass diagnosed and not worry about the amio for now.by Carey - AFIBBERS FORUM
Quotesmackman Not trying to be negative but Multaq did not work for me. That's what I told Natale, and he put me on it anyway following my ablation. And it worked. Multaq is a "mild" antiarrhythmic that probably won't help with a hard core case of afib, but it is good at calming down an inflamed heart following ablations, and it's worth at least trying for almost anyby Carey - AFIBBERS FORUM
Quotecolindo I wonder under the circumstance if Shannon would have had a TIA no mater what anti-coagulate he used? I would like to know what the Romanian lady was taking, if anything? I have never seen any comparisons between nattokinase and other anti-coagulates. I think it's likely he would not have suffered the TIA. Hindsight, of course, and impossible to know for sure, but the effeby Carey - AFIBBERS FORUM
Just so readers are clear on this, there is absolutely no connection between atrial fibrillation and ventricular fibrillation despite the similar names. The former is a manageable condition that will not reduce your lifespan as long as you receive proper medical care. The latter is an immediately fatal condition also known as sudden cardiac arrest. Afib does not cause or promote ventricular fiby Carey - AFIBBERS FORUM
QuoteIce Man Thanks. Never heard of Multaq, but would its side effects be worse than Metoprolol's? It tends to have few if any side effects. I never noticed any.by Carey - AFIBBERS FORUM
I'm unclear on whether you're currently taking the Multaq or not. If you are, I'd go ahead and stop the metoprolol unless you flip back into afib and your rate is over 100. If you're not, then I would stop the metoprolol and start the Multaq on the same day. If it works, then no need for the metoprolol (unless you go back into afib > 100). I've been on Multaq twiceby Carey - AFIBBERS FORUM
Persistent afib is definitely more difficult to ablate -- significantly more difficult, as shown by the success rates for all EPs, which is about 50% at best, but realistically more like 40%. Top EPs achieve much higher success rates, but I can count the number of them in the US on two hands, and worldwide maybe three hands. Since you're asymptomatic, currently in normal sinus rhythm (NSby Carey - AFIBBERS FORUM
A resting HR in the 50s and even 40s is nothing to be afraid of as long as you're not lightheaded or dizzy. Multaq has only a very mild HR lowering effect, so it's not a problem with the metoprolol. But if it works for you, there's really no reason to be taking the metoprolol at all. What you can do is reserve the metoprolol for times you're actually in afib, but for that you&by Carey - AFIBBERS FORUM
No, it's not. I've never heard of anyone experiencing back pain from Eliquis and it's not listed as a known side effect. Considering that virtually every single thing anyone ever reports about a drug gets listed as a side effect whether true or not, the fact that it's not listed is a pretty good indication Eliquis isn't the cause. I can't even imagine a mechanism byby Carey - AFIBBERS FORUM
Not all PMs and monitors use wifi, and if they don't use wifi they can't communicate with a phone.by Carey - AFIBBERS FORUM
Sure, it's perfectly normal to be in afib during an ablation or to go into it during the procedure. The fact that the afib stopped when he ablated to next area is proof that he ablated a confirmed source of afib. There's absolutely nothing about those notes that should cause you concern.by Carey - AFIBBERS FORUM
[email protected] Medtronic told me to use the (fob) that came along with the device. I think if your phone could do that they would have told you, or it would at least be mentioned in the instructions. I doubt it's even possible for a phone to interact with the device. How would it? Bluetooth signals don't penetrate the human body, and near field communications probably wouby Carey - AFIBBERS FORUM
Hi Neal, welcome to the forum. I found your post added to an old thread about someone else, so I made it into a new thread all its own. This is an interesting and very important question.by Carey - AFIBBERS FORUM
QuoteBrian_og The math makes sense but that part just seems weird. But is it really? Last I checked stroke is the #3 cause of death among the elderly.by Carey - AFIBBERS FORUM