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Quotetibbar I meant to type that I have had no episodes in the last 2 years (the pacemaker records them). In that case, then a trial of reducing or eliminating flec may make some sense. Because flec can lose its effectiveness over time, it makes sense to use the lowest possible dose that is effective. If you could go to no flec and use it only on demand if/when you have an episby GeorgeN - AFIBBERS FORUM
Hi tibbar, Flec can lose its efficacy over time, which may be happening to you. Also, your disease may be progressing, making it more difficult for flec to control the afib. In any case, a chat with your EP about your options (different med, or perhaps ablation) may be in order. It seems was initially effective, with your episode frequency dropping from ~ once a week to ~ 1-4 episodes a yby GeorgeN - AFIBBERS FORUM
Quotetsco I guess im looking for input …am I imagining that the Pot Cl is helping? Are these dangerous? I know you have to be careful with potassium but is this dosing insignificant? or significant? I dont want to kill myself so I just wonder if this is a wrong move? I also take magnesium every night Hi Tim, The issue with any electrolyte being dangerous is the status of your kidneys.by GeorgeN - AFIBBERS FORUM
QuoteStarwarsfan I’ve taken at the entire time I’ve had a fib, since 2016. Who else has thoughts on this? George? Afib started in 2004. My first omega3 index test was in 2015 and was around 3%. With my genetics my doc likes the index > 10%. Hence I've taken material quantities since. I've run it as high as 17% and not noticed any afib impact.by GeorgeN - AFIBBERS FORUM
Quotesusan.d Does my wavy zigzag resemble this? I don't think so, from your wiki link, "During ventricular fibrillation, cardiac output drops to zero, and, unless remedied promptly, death usually ensues within minutes." Since you wrote this and went back to chopping veggies.... Looks like artefact to me.by GeorgeN - AFIBBERS FORUM
Also these issues of the afib report Silent Cerebral Lesions: Association with both AFIB and Ablations Association between AFIB and Silent Cerebral Infarctionsby GeorgeN - AFIBBERS FORUM
Betty Lou, See Shannon’s posts on the topic of long duration afib and the association with ischemia and dementia:by GeorgeN - AFIBBERS FORUM
FWIW, here is Google Scholar search on the topic:by GeorgeN - AFIBBERS FORUM
QuoteRitva Apparently Flec stays in your system up to four days. The quoted half life of flec is 12 hours. This could vary by individual. So after 24 hours, the flecainide in your system would be half of a half or 25%. This is very much a generalization. "5.2 Pharmacokinetic properties Source Following oral administration, the absorption of flecainide acetate is almost complete (9by GeorgeN - AFIBBERS FORUM
QuotePoppino Dead hangs i recently learned from Rich here on this site. An ortho wrote a book on it Yes, this is the book: If it is biceps, then hanging may not be the best approach. However trigger points might be useful.by GeorgeN - AFIBBERS FORUM
Tom, Possibly trigger point issues? Book on topic here: Also online reference: If not familiar, trigger point concept - localized muscle spasms that can refer pain elsewhere. In the book above, they teach how to look for trigger points as well as releasing them with massage or pressure. A couple of other release techniques I've learned : 1) is to put an ice pack over the trigby GeorgeN - AFIBBERS FORUM
300mg, 200mg for those less than 154# (70 kg) See: See Methods section {edit} sorry about the ng instead of mg typo (which I corrected)- I was on my phone & my old eyes didn't notice!by GeorgeN - AFIBBERS FORUM
QuotePoppino Ill assume he never got a Watchman? Correct, he did not get a Watchman. As I recall, his index ablation was very extensive. Shannon said my friend's time in the procedure was 2nd only to his own. My buddy was long standing persistent starting this journey. My friend lives in Houston. He was like, why are you telling me to go to Austin, we've got great docs heby GeorgeN - AFIBBERS FORUM
Hi Tom, My buddy had two Natale ablations, including LAA isolation, 6 or 7 years ago. Initially he was in the "red" zone for getting off OAC, but not too far away. After a couple of years ago he improved into the "green" zone and is off all meds. I don't know that he did anything specifically to improve, just luck, I guess. Other than afib, he's very healthy.by GeorgeN - AFIBBERS FORUM
QuoteHowenow Has anyone else experienced this? It does show up on this page as a rare flec side effect: "unusual tiredness or weakness." It is also a less common side effect for Diltiazem. I've used flec on-demand to convert for 17 years but only one month of that time was chronic usage. I've not experienced this issue. As I take a loading dose to convert, I usually wby GeorgeN - AFIBBERS FORUM
QuoteSkford As I understand it the trial is randomized so even if I sign up I may not get the PFA. What's the current thinking on PFA? Here is a search on our site on the topic: One of our members is in the trial and has posted updates. The search will pick those up. You could also PM that member and get a more first hand report. So far, my impression from afar is very positive.by GeorgeN - AFIBBERS FORUM
Quotecolindo Just to turn things around a bit, You seem to be inferring that nattokinase is inferior to Eliquis, that may be so but where is the proof, who can say for sure? can you? The problem isn't that we know nattokinase is inferior to the prescribed anticoagulants, the problem is that we don't know. We know the efficacy of the prescribed meds and risking a stroke when you doby GeorgeN - AFIBBERS FORUM
If I had more than a minimal (<=2) CHA₂DS₂-VASc, I personally would not rely on NK. Aspirin is not a preferred choice for afib either. Not that I think NK is bad, I just would not rely on it. I recall a number of years ago, when Shannon had a TIA, he was on 300 mg /day in three 100mg divided doses.by GeorgeN - AFIBBERS FORUM
If you are on an anticoagulation med, then it should not matter. Checking your pulse once or twice a day would be sufficient to see if you were in afib and start taking metoprolol for rate control. A high rate for a part of a day would not be an issue for cardiomyopathy, the other big risk for afib. Rates have to be elevated (> 100 or 110) for longer for this to be an issue.by GeorgeN - AFIBBERS FORUM
QuotePoppino George he could buy a Kardia to monitor his afib? I have one but like Carey said : put it away! Due to my almost ocd pulse ckg. My rates are always high so i dont need Kardia to know im in a bad rythm. But the slow asymptotic afib is different Sure, but if he goes off the metoprolol, then his BP monitor works also as the rate will be higher.by GeorgeN - AFIBBERS FORUM
Susan, If this is a new result in this blood test, it is unlikely primary carnitine deficiency. Probably not too many vfibbers here as it is usually fatal absent very fast access to a defib machine. I'd ask your smart primary guy to help you figure out the underlying cause.by GeorgeN - AFIBBERS FORUM
Here is my take. When I first was diagnosed with afib, 17 years ago, one of the things I heard was that afib was "the hemorrhoids of cardiology." As you know, the two big risks of afib are stroke (which the Eliquis mitigates) and cardiomyopathy leading to heart failure from extended rates over 100-110 BPM. The metoprolol will mitigate this. So the doc has done their job to keepby GeorgeN - AFIBBERS FORUM
I concur with Carey. If you are in NSR, likely no need for metoprolol. If you are in afib, then yes, you'd want to keep your rate < 100. If you have an adrenergic trigger then there is a rationale that having metoprolol act as a "governor" on your heart rate might keep you in rhythm. Since you just saw you were in afib from your BP monitor, I'm sure you don't haveby GeorgeN - AFIBBERS FORUM
QuoteMarkL Up until the past couple of months I simply refused to believe it could be the amount of biking I was doing. That was a combination of my ignorance of the Afib topic and the fact that my endurance had actually increased AND the fact that these episodes have NEVER occurred the night of a mountain bike ride earlier in the day. Turns out any of the contributing factors can have a cumulby GeorgeN - AFIBBERS FORUM
The purpose of the Metoprolol is to reduce your heart rate during afib. If your heart rate is going to 167 during afib, it clearly isn't keeping your heart rate low. As you may know, there are two big risks with afib. One is a stroke caused by a clot (so you may have been prescribed an anticoagulant med) The other is having high heart rates for an extended period of time (>100). Thisby GeorgeN - AFIBBERS FORUM
Mark, Some more observations. - I learned early on that competing was a bad plan for me (whether with myself or others). My son always wanted me to go to CrossFit class with him and I would not do it as I knew I'd want to complete with him and the others - which would be counterproductive for my afib. - One of the things I do that could be like your bike riding is alpine skiingby GeorgeN - AFIBBERS FORUM
QuotePompon Shouldn't we be cautious with potassium? In which case can it be dangerous? Mg is harmless if we don't have bad kidneys. But what with K? And what may be considered excess? Edit: I've yet tried K, without any effect, but quantities were moderate... Bad kidneys are also an issue with potassium as are consuming some meds. Healthy kidneys will excrete a bolus of poby GeorgeN - AFIBBERS FORUM
Hi Mark, I'll relate my experience. I'm 66 and my path to afib 17 years ago was chronic fitness. My first episode was about two days after a training run on a 14,000' peak - woke up with it. After two months of getting afib every 10-14 days, lasting 6-9 hours, I had an episode that would not convert. My EP suggested staying out of rhythm as my afib heart rate was low. Iby GeorgeN - AFIBBERS FORUM
QuotePoppino Many yrs ago Hans had on this site a recipe called PAC Tamer. I don’t remember its ingredients Here it is:by GeorgeN - AFIBBERS FORUM
QuoteSam3726 I have to remember to use salt. I add 1 tsp of sea salt into my liter of water with my potassium citrate. Yes, I also eat no processed foods.by GeorgeN - AFIBBERS FORUM